Deadly Blood Clots Are Being Caused by COVID-19
The Times of INDIA (that’s how far we had to go to find this story) is reporting an alarming increase in the percentage of COVID-19 patients who are suffering strokes, heart attacks, and other ailments because of unusual blood clotting attributed to the Sars-2 virus. In many cases, these patients have NO predisposition toward these ailments.
According to the Times of India article, doctors in different specialties at New York’s Mt. Sinai Hospital started to compare notes. Nephrologists noticed that kidney catheters were getting clogged with clots. Pulmonologists found that parts of the lungs of COVID-19 infected patients weren’t getting any blood flow at all. There was a surge of strokes and heart attacks noticed among COVID-19 patients. Strokes result from blood clots in the brain, as do many heart attacks.
Doctors have already found that by the time many COVID-19 patients get to the emergency rooms, their oxygen levels are already down as much as 50% from normal, indicating that their blood wasn’t being oxygenated by their lungs. (Technically, a 50% O2 level is indicative of advanced hypoxia.) It was thought that this was because the COVID-19 lays down a barrier over the permeable membranes of the lungs through which oxygen transfers into the blood stream while CO2 is transferred out.
Well, it turns out that the CO2 WAS being transferred out through the lungs and exhaled, but the oxygen wasn’t getting through the air/blood barrier. It is the build-up of CO2 in the body that causes perceptible physiological warning signs of oxygen starvation, not the shortage of oxygen itself in the body, causing shortness of breath, then heavy breathing, coughing and the other symptoms of pulmonary distress.
Why, then, should this thick mucus-like layer of COVID-19 allow CO2 to pass out while preventing O2 from passing in?
Oxygen molecules have a kinetic diameter of 346 pn. Carbon dioxide molecules have a kinetic diameter of 330 pn. It is difficult to believe that a delta of just 16 pn would allow a CO2 molecule to pass through a barrier that would prevent an oxygen molecules from making the same passage but in the other direction. Possible, but unlikely.
What is far more likely is that blood thickened by minuscule blood clots formed by COVID-19 “spores” attaching themselves to red blood cells reduces the surface area of the red blood cells that is available for oxygenation, or that the clotted blood cells lose their ability to absorb oxygen coming through the lungs, thus accounting for the low O2 levels.
Other symptoms of COVID-19 infection that could be the result of blood clotting includes the sudden rash of rashes on the bodies of COVID-19 patients which may be the result of tiny blood clots in the blood stream. Some of these larger rashes have resulted in amputations of limbs when the clots cut off blood circulation to fingers, toes, arms, and legs. Headaches and muscle and body aches can also be attributed to the thickening of the blood by COVID-19 blood clots.
The result of these observations has been the development of a protocol calling for the administration of high doses of blood-thinning drugs to COVID-19 patients BEFORE the patient’s blood begins to clot.
This, in turn, raises questions about the advice with respect to advisability of taking low-dose aspirin as a preventive measure for patients who have not had heart attacks or strokes. Now, older patients who have not had heart attacks or strokes are being told not to continue a low-dose aspirin regimen on the grounds that the blood-thinning effects of the aspirin was causing internal bleeding in some patients.
Right now might be a good time reconsider that advice and start taking low doses of aspirin again to counteract the clotting effects of the COVID-19 virus…and maybe upping those doses a bit.
There is absolutely NO clinical research supporting this recommendation. It is simply common sense. If the virus is devastating the human body primarily by causing blood clotting, taking a drug that thins out the blood may or may not stop the progress of the disease…but it probably isn’t going to hurt you, one way or the other (unless, again, you have been told you have a clotting factor problem.)
Aspirin is not without its dangers, especially for children, but, for adults, the dangers posed by aspirin are exactly the same effects that might combat the effects of the virus.
This is by no means a cure, nor is it even a proven prophylactic measure but, if doctors are treating COVID-19 patients with blood thinners BECAUSE of the prevalence of clotting connected issues, doesn’t it make sense to do what you can (unless you have been advised otherwise by your physician) to thin out your blood just a little bit every day?.
I really want feedback on this observation. I am putting it out because it seems important, as did my article about checking your O2 levels on a regular basis, but I don’t want to be one of those “experts” who are really only experts at attracting attention to themselves.
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